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[39] In animals, sodium valproate raises cerebral and cerebellar levels of GABA, possibly by inhibiting GABA degradative enzymes, such as GABA transaminase, succinate-semialdehyde dehydrogenase and by inhibiting the re-uptake of GABA by neuronal cells

elegans , and aged mice with trigonelline resulted in increased NAD + levels and enhanced mitochondrial function through the PreissHandler pathway, and improved muscle aging phenotypes 124

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Collectively, treatment with sulforaphane rescued rotenone induced oxidative damage by activating the Nrf2 pathway